<?xml version="1.0" encoding="UTF-8"?>
<rss xmlns:dc="http://purl.org/dc/elements/1.1/" version="2.0">
  <channel>
    <title>NOPR Collection:</title>
    <link>http://nopr.niscpr.res.in/handle/123456789/23706</link>
    <description />
    <pubDate>Fri, 09 Oct 2026 09:57:16 GMT</pubDate>
    <dc:date>2026-10-09T09:57:16Z</dc:date>
    <item>
      <title>Susceptibility of diamond back moth, &lt;i&gt;Plutella xylostella &lt;/i&gt;(L) to entomopathogenic nematodes</title>
      <link>http://nopr.niscpr.res.in/handle/123456789/24092</link>
      <description>Title: Susceptibility of diamond back moth, &lt;i&gt;Plutella xylostella &lt;/i&gt;(L) to entomopathogenic nematodes
Authors: Shinde, Shilpa; Singh, N.P.
Abstract: Eight entomopathogenic nematode species &lt;i&gt;/&#xD;
&lt;/i&gt;strains, &lt;i&gt;Steinernema glaseri &lt;/i&gt;(steiner), &lt;i&gt;S. carpocapsae &lt;/i&gt;(Weiser),&#xD;
&lt;i&gt;S. feltiae &lt;/i&gt;(Filipjev), &lt;i&gt;Steinernema&lt;/i&gt; sp. Ecomax strain, &lt;i&gt;Heterorhabditis&#xD;
bacteriophora &lt;/i&gt;(Pioner), &lt;i&gt;Heterorhabditis &lt;/i&gt;sp. Ecomax strain, two&lt;i&gt; &lt;/i&gt;locally&#xD;
isolated strains called as JFC and TFC were tested against the final instar larvae&#xD;
of diamond back moth, &lt;i&gt;Plutella&lt;/i&gt;&#xD;
&#xD;
&lt;i&gt;xylostella &lt;/i&gt;(L.). All nematodes were&#xD;
found pathogenic. However, &lt;i&gt;H. bacteriophora &lt;/i&gt;was adjudged the most pathogenic&#xD;
amongst the test nematodes on the basis of LD&lt;sub&gt;50&lt;/sub&gt; (9.16 IJS/larva), LT&lt;sub&gt;50&lt;/sub&gt;&#xD;
(43.26 hr), Lex T&lt;sub&gt;50&lt;/sub&gt; (3.24 hr) and the propagation&#xD;
potential (average of 271.42 IJS/mg) on the host body weight.
Page(s): 956-959</description>
      <pubDate>Fri, 01 Sep 2000 00:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://nopr.niscpr.res.in/handle/123456789/24092</guid>
      <dc:date>2000-09-01T00:00:00Z</dc:date>
    </item>
    <item>
      <title>RAPD markers linked to brown planthopper &lt;i&gt;Nilaparvatha lugens &lt;/i&gt;resistance locus in rice</title>
      <link>http://nopr.niscpr.res.in/handle/123456789/24090</link>
      <description>Title: RAPD markers linked to brown planthopper &lt;i&gt;Nilaparvatha lugens &lt;/i&gt;resistance locus in rice
Authors: Amudha, J; SreeRangasamy, S R; Jayapragasam, M; Balasubramani, G
Abstract: Brown plant hopper, a major pest in rice causes&#xD;
‘‘hopper burn’’ in the field. The resistance gene for brown planthopper was mapped&#xD;
by using 20 recombinant inbred lines (RIL's) derived from a cross between resistant&#xD;
line &lt;i&gt;Oryza. officinalis&lt;/i&gt; derivative (IR 54742-2-21-12-17-6) and a susceptible&#xD;
rice cultivar ASD 16 using bulked segregant analysis. On an average&#xD;
&#xD;
of 4 loci were amplified and two RAPD primers&#xD;
amplified loci that co-segregated with resistance/susceptibility. The segregating&#xD;
RAPD loci were mapped using Mapmaker programme into 13 groups. The expected and&#xD;
the 95% confidence level were found to be 15.2 and 47.7 eM respectively, confirming&#xD;
the location of the brown planthopper resistant gene on&#xD;
&#xD;
the region of chromosome 4 . These RAPD&#xD;
markers will accelerate breeding programme for brown planthopper resistance.
Page(s): 951-955</description>
      <pubDate>Fri, 01 Sep 2000 00:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://nopr.niscpr.res.in/handle/123456789/24090</guid>
      <dc:date>2000-09-01T00:00:00Z</dc:date>
    </item>
    <item>
      <title>Aluminium related changes in brain histology: Protection by calcium and nifedipine</title>
      <link>http://nopr.niscpr.res.in/handle/123456789/24088</link>
      <description>Title: Aluminium related changes in brain histology: Protection by calcium and nifedipine
Authors: Basu, Srabanti; Gupta, Romita Das; Chaudhuri, Aditi Nag
Abstract: Aluminium (Al; 50 mg AlCl&lt;sub&gt;3&lt;/sub&gt;/kg&#xD;
body wt/day) treatment caused a marked change in histological picture of normal&#xD;
brain as indicated by an increased number of vacuolated spaces. These changes&#xD;
returned to normal partially by simultaneous treatment with nifedipine (0 .7&#xD;
mg/kg body wt/day) and completely by similar treatment with 50 ppm calcium (CaCl&lt;sub&gt;2&lt;/sub&gt;&#xD;
; 12.5 mg/kg body wt./day). Neither nifedipine nor calcium treatment alone&#xD;
altered the normal histological condition. The histological changes could not&#xD;
be correlated with the decrease in calcineurin activities in brain as nifedipine&#xD;
decreases calcineurin activity without any histological changes. Hence the&#xD;
histological changes may be considered as specific for Al&#xD;
&#xD;
and not due to a general decrease in&#xD;
calcineurin activity.
Page(s): 948-950</description>
      <pubDate>Fri, 01 Sep 2000 00:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://nopr.niscpr.res.in/handle/123456789/24088</guid>
      <dc:date>2000-09-01T00:00:00Z</dc:date>
    </item>
    <item>
      <title>Effect of &lt;i&gt;Emblica officinalis &lt;/i&gt;tannoids on a rat model of tardive dyskinesia</title>
      <link>http://nopr.niscpr.res.in/handle/123456789/24086</link>
      <description>Title: Effect of &lt;i&gt;Emblica officinalis &lt;/i&gt;tannoids on a rat model of tardive dyskinesia
Authors: Bhattacharya, Salil K.; Bhattacharya, Dipankar; Muruganandam, A.V.
Abstract: Effect of active tannoid principles of &lt;i&gt;E&lt;/i&gt;.&#xD;
&lt;i&gt;officinalis&lt;/i&gt;,&lt;i&gt; &lt;/i&gt;comprising of emblicanin A (37%), emblicanin B (33%),&#xD;
punigluconin (12%) and pedunculagin (14%), was investigated on a rat model of tardive&#xD;
dyskinesia (TD) induced by once daily administration of haloperidol (1.5 mg/kg,&#xD;
ip) for 28 days. Involuntary oro facial movements (chewing movements, buccal&#xD;
&#xD;
tremors and tongue prolusion) were assessed&#xD;
as TO parameters. The tannoid principles of &lt;i&gt;£.officina/is &lt;/i&gt;(EOT) wery&#xD;
administered concomitantly with haloperidol in the doses of 10, 20 and 50&#xD;
mg/kg, po, for 28 days. Sodium valproate (200 mg/kg, po), a Gaba-mimetic agent,&#xD;
and vitamin E (400 mg/kg, po), an antioxidant, were used as the standard drugs&#xD;
and administered for the same period. EOT induced a dose-related inhibition of&#xD;
all the three TD parameters assessed, as did vitamin E. The effect of sodium&#xD;
valproate remained statistically insignificant. The results suggest that EOT&#xD;
exerts a prophylactive effect against neuroleptic-induced TD which is likely to&#xD;
be due to its earlier reported antioxidant effects in rat brain areas, including&#xD;
striatum.
Page(s): 945-947</description>
      <pubDate>Fri, 01 Sep 2000 00:00:00 GMT</pubDate>
      <guid isPermaLink="false">http://nopr.niscpr.res.in/handle/123456789/24086</guid>
      <dc:date>2000-09-01T00:00:00Z</dc:date>
    </item>
  </channel>
</rss>

