Please use this identifier to cite or link to this item: http://nopr.niscpr.res.in/handle/123456789/23633
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dc.contributor.authorRao, M Rajeswara-
dc.contributor.authorKumari, K Vijaya-
dc.contributor.authorDass, P Jacob-
dc.contributor.authorVijaya, P-
dc.contributor.authorBharathi, P Vijaya-
dc.date.accessioned2013-11-18T04:49:15Z-
dc.date.available2013-11-18T04:49:15Z-
dc.date.issued2001-01-
dc.identifier.issn0975-1009 (Online); 0019-5189 (Print)-
dc.identifier.urihttp://hdl.handle.net/123456789/23633-
dc.description68-69en_US
dc.description.abstractEffect of fructose 1 ,6-diphosphate (FOP) and carbon tetrachloride (CCl4) were studied individually and in combination on rat endothelial (ET) and smooth muscle cell (SMC) nitric oxide synthase (NOS) activities in vivo, inhibition of ET and SMC NOS activity in CCl4 treated rats was reversed in FDP+CCl4 treated animals. Cellular based NOS activity was significantly increased in FOP treated group of rats when compared to non treated controls. The results suggest a significant increase in NOS in rats treated with a combination of FOP+ CCl4 thus overcoming the suppression of NOS exposed to CCl4 alone. en_US
dc.language.isoen_USen_US
dc.publisherNISCAIR-CSIR, Indiaen_US
dc.rights CC Attribution-Noncommercial-No Derivative Works 2.5 Indiaen_US
dc.sourceIJEB Vol.39(01) [January 2001]en_US
dc.titleFructose, 1,6-diphosphate mitigates CCl4 suppressed nitric oxide synthase activity in rats en_US
dc.typeArticleen_US
Appears in Collections:IJEB Vol.39(01) [January 2001]

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