Please use this identifier to cite or link to this item: http://nopr.niscpr.res.in/handle/123456789/24014
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dc.contributor.authorGurib, F B H-
dc.contributor.authorSubratty, A H-
dc.date.accessioned2013-11-21T11:20:20Z-
dc.date.available2013-11-21T11:20:20Z-
dc.date.issued2001-09-
dc.identifier.issn0975-1009 (Online); 0019-5189 (Print)-
dc.identifier.urihttp://hdl.handle.net/123456789/24014-
dc.description929-932en_US
dc.description.abstractPossible pharmacological effects of N-α-tosyl L-arginine methyl ester [TAME] were studied on rat aorta strips in vitro. Results showed that [TAME]-esterase was an endothelium dependent component that involved a nitric oxide cyclic-GMP mediated pathway. Furthermore, during activation of Kinin-Kallikrein system, TAME-esterase induced contractions involve degradation of kinins by kininases. en_US
dc.language.isoen_USen_US
dc.publisherNISCAIR-CSIR, Indiaen_US
dc.rights CC Attribution-Noncommercial-No Derivative Works 2.5 Indiaen_US
dc.sourceIJEB Vol.39(09) [September 2001]en_US
dc.titleExperimental evidence for a non-renin mediated pathway during TAME-esterase induced contractions in rat aorta in vitro en_US
dc.typeArticleen_US
Appears in Collections:IJEB Vol.39(09) [September 2001]

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